OCD of the Elbow

Osteochondritis dissecans (OCD) is a musculoskeletal problem occurring primarily in the maturing skeleton. Early descriptions of the problem came from the surgeons who opened the knee joint looking for the cause of catching and locking symptoms. In these first cases, large loose OCD fragments were found and removed, making the patients significantly better in the short run. These individuals, however, then went on to have significant problems and poor outcomes. Since that time improved imaging ability has advanced our understanding and ability to treat the problem. However, there are still many unanswered questions with regards to both the etiology and the best treatment options for OCD’s when diagnosed.

The most common location for OCD’s is within the maturing knee along the lateral border of the medial femoral condyle.(Fig. 1) The overall incidence of these lesions has been reported between 15 and 29/100,000[1][2] with a significant number of lesions being asymptomatic, and a 2:1 prevalence for boys as compared to girls. This incidence is most certainly rising and age at presentation is decreasing. This is due to both the increased awareness of the disease as well as the increasing involvement of young athletes in competitive athletics.[3][4] This same increasing incidence of OCD’s is found in other joints such as the ankle, elbow, shoulder, and hip. One of these – the elbow – in particular has a rising incidence as well as increased interest and investigation of.

The recent increased interest in elbow OCD’s likely has occurred due to the increased competitive involvement of young athletes as mentioned above. Increased year-round participation in gymnastics and baseball as well as the advent and use of high resolution MRI’s have increased the awareness and diagnosis of the problem. The increased awareness also is due in part to the better understanding of the diagnosis and the differences between capitellar OCD’s osteonecrosis, osteochondrosis, hereditary epiphyseal dysplasia, Little Leaguer’s elbow, and most importantly, Panner’s Disease with which it is often confused. [5] Panner’s disease presents very similarly to a capitellar OCD but is a self-limited disease which usually occurs in patients under the age of 10. (Fig. 2 and 3)[6][7] Elbow OCD’s, which also have been reported in the trochlea, radial head, and the olecranon, usually are seen in slightly older patients, usually in athletes involved in upper extremity dominant sports. These sports place repetitive weight bearing stresses on the elbow – such as gymnastics – or repetitive demands – such as baseball, softball, and javelin throwers. These repetitive stress sports lead to early changes in the articular cartilage and stress reaction of the subchondral bone. If allowed to continue before the stress reaction ‘heals’, these stresses are believed to be one of the causes of OCD’s. As opposed to Panner’s Disease where good outcomes are relatively common, the outcome is not always good for OCD’s with the prognosis and outcome dependent on the patient age, location and severity at the time of diagnosis.[8][9][10]


Many causes of OCD’s are suspected and often the true etiology is unknown. Originally proposed to be inflammatory in origin, Konig’s 1887 theory, which he referred to as osteochondritis dissecans, seems not to be correct. The etiology of knee OCD’s is sometimes reported to be secondary to micro or macro-trauma. In the elbow especially given the high correlation between these lesions and the involvement in baseball and gymnastics, the etiology of elbow OCD’s seems even more likely to be repetitive trauma in nature. Given the tenuous blood supply of the capitellum demonstrated by Haraldsson it seems more likely in this case as opposed to the knee that the repetitive nature of overhead athletes and gymnasts placing exuberant stresses on the elbow that microtrauma is at least partially to blame etiologically.[11][5]


Athletes with capitellar OCD’s often have diffuse, non-specific complaints that center around activity. They complain of mild, inflammatory-like symptoms after exercise. Rest and anti-inflammatory treatment often is effective for their symptoms early in the disease process which is often the cause that early diagnosis of these lesions is not common. Athletes will often wait to seek medical advice until they feel catching or locking and sharp pain during their activity. At this point their performance is starting to suffer and they may have an effusion as well as a loss of terminal extension. Occasionally if the lesion is advanced where a loose fragment exists, a noticeable click or even locking intermittently may be present.

Physical exam centers on looking for evidence of chondral or osseous irregularities. Range-of-motion including supination and pronation should be checked and followed over time as the loss of extension especially is a bad omen. Occasionally an effusion can be felt as well as an area of focal tenderness. Both of these when present, are most commonly found in the posterolateral corner of the elbow – the so-called soft spot. While difficult, a ligamentous exam should also be performed as laxity in overhead athletes can lead to many of the symptoms that OCD’s can.

Standard elbow x-rays should be taken including oblique views of the elbow – radial head views – to determine if early necrosis of the capitellum exists as well as fragmentation of the lesion. Occasionally a CT scan will be helpful to better define the subchondral bone condition. The hallmark for imaging and further diagnosis is the MRI. The MRI allows an evaluation both of the articular cartilage and underlying bone with determination of the separation of the bone island from the native bone if it has occurred. High field MRI’s with cartilage sequences are recommended and the increasing availability of 3.0 tesla MRI’s is very helpful in the evaluation.

Non-operative Treatment

Non-operative treatment when the OCD is diagnosed at an early stage can be successful. Reduction or elimination of all stress on the capitellum for a period of at least 6 weeks allows the subchondral bone to stabilize and support the overlying cartilage. After this initial rest period, a slow return of daily activities and then athletic activities can be allowed. Most authors suggest that this time-line is perhaps as long as 6 months in order to allow complete healing. [9] Though not well established in the elbow, it is thought that when the MRI does not show separation of the fragment or synovial fluid within the OCD, that rest will result in a full functional recovery in 50% or more of the cases. This is certainly true in the treatment of knee OCD’s and the results are further improved in younger, pre-pubescent athletes with wide-open physes. [12]

When rest is difficult to accomplish, bracing, use of a sling, or even a period of casting is utilized. Return to activities is appropriate only after there is complete resolution of symptoms and the athlete has regained full range-of-motion and strength not only about the elbow but of the shoulder and shoulder girdle. [13]

Operative Treatment

When non-operative means are unsuccessful or the evaluation determines that a fragment is loose, operative treatment is appropriate. This usually starts with an arthroscopy of both the anterior and posterior compartments of the elbow. Small, loose fragments are removed and synovitis is treated as need be. The OCD itself is then treated. Appropriate treatment is determined both by future demands of the patient as well as the pre-operative symptoms, appearance on imaging studies, and arthroscopic appearance of the lesion. Many options exist though few controlled trials exist to help determine the best option. [14]
In cases when the lesion is an early stage of development, most options center on helping the subchondral bone to ‘heal’. When there is no break in the articular surface drilling of the reactive bone has been reported to achieve excellent results. The drilling can be done either antegrade or retrograde depending on the accessibility of the lesion and its extent. The elbow must be allowed to rest after the drilling so that the bone stabilizes before weight bearing or repetitive activities are instituted. When done early in the disease process, these lesions do very well and a full return to pre-morbid levels of activity is normally achieved. [15]

When the lesion is further along the continuum and has articular cartilage fissures and/or the OCD fragment appears separating or is separating from the native bone, fixation of the fragment is recommended.(Fig. 4) This can be achieved with a myriad of devices including bioabsorbable pins or screws, metal screws, bone dowels, or direct suturing of the fragment.[14][16] Similarly, if the fragment is loose and synovial fluid has seeped between the fragment and the native bone, preparation of the bed and often bone grafting is necessary. Though possible arthroscopically, making an arthrotomy to perform open bone grafting and repair is usually performed. [17]

When the fragment is loose, unable to be fixed or has little to no bone on it, excision of the fragment and meticulous debridement is performed to remove fibro cartilage to enhance the chance for the lesion to fill in. This does seem to provide good pain relief, at least in the short run, and improvement of function when the lesion is contained.(Fig. 5)[18] However, when the lesion does not have intact edges or the lesion wraps around the edge of the capitellum, poorer results from debridement are seen. In these cases, some authors have reported good results with osteochondral autografting or allografting procedures. Long term follow-up of these procedures are not yet known. [19][20]


The presentation of capitellar OCD’s is variable and the duration of symptoms prior to presenting to the physician’s office is often quite long. After diagnosis, rest is the mainstay of treatment and provided the lesion is not advanced along the continuum of the disease, good results can be anticipated. Unfortunately, however, these lesions often present late in their course and often require surgical intervention. Recurrent clicking or locking of the elbow often indicates that the OCD fragment is loose or free within the joint. In this situation as well as when x-ray or MRI imaging determine that the fragment is unstable rarely have good outcomes when treated non-operatively. (Fig. 6) Surgery in these cases when it is possible to stabilize the fragment and allow it to heal has good results. However, when the fragment is not fixable, while surgical treatment may provide symptomatic relief, results are variable and the long-term results are unknown. Elbow capitellar OCD lesions are being diagnosed more often. This is perhaps due to the increased specialization of young athletes or the increased ability to diagnose them. In either case, when annoying, aching elbow pain exists in young athletes – certainly when a history of locking exists – examination to determine the presence or not of an OCD lesion is appropriate. When diagnosed in the early stages, treatment is commonly successful with a full return to daily and sporting activities.

Fig 1. MRI of a significant medial femoral condyle OCD.

Fig.1 MRI of a significant medial femoral condyle OCD.

Fig 2. X-ray of young gymnast with an OCD of the humeral capitellum.

Fig.2 X-ray of young gymnast with an OCD of the humeral capitellum.

Fig 3. MRI scan of the same patient as seen in Fig. 2 with more extensive involvement of the humeral capitellum.

Fig.3 MRI scan of the same patient as seen in Fig. 2 with more extensive involvement of the humeral capitellum.

Fig 4. Initial arthroscopic appearance of a humeral capitellar OCD. Fragment is obviously loose but grossly in place.

Fig.4 Initial arthroscopic appearance of a humeral capitellar OCD. Fragment is obviously loose but grossly in place.

Fig 5. Arthroscopic appearance of significant capitellar OCD. View is from the posterior compartment with radial head at the top of the picture. This is a contained lesion which did well after removal of the fragment.

Fig.5 Arthroscopic appearance of significant capitellar OCD. View is from the posterior compartment with radial head at the top of the picture. This is a contained lesion which did well after removal of the fragment.

Fig 6. Humeral capitellar OCD with minimal bone involvement.

Fig.6 Humeral capitellar OCD with minimal bone involvement.

Open References

1. Hugston, J., P. Hergenroeder, and B. Courtenay, Osteochondritisdissecans of the femoral condyles. JBJS, 1984. 66: p. 1340-1348.
2. Linden, B., The incidence of osteochondritis dissecans in the condyles of the femur. Acta Orthop Scand, 1976. 47(6): p. 664-7.
3. Cahill, B., Osteochondritis dissecans of the knee: treatment of juvenile and adult forms. JAAOS, 1995. 3: p. 237-247.
4. Kocher, M.S., et al., Management of osteochondritis dissecans of the knee: Current Concepts Review. AJSM, 2006. 34: p. 1181.
5. Baker, C.L., A.A. Romeo, and C.J. Baker, Osteochondritis dissecans of the capitellum. AJSM, 2010. Preview Jan 23, 2010: p. 1-12.
6. Bradley, J.P. and R.S. Petrie, Osteochondritis dissecans of the humeral capitellum. Diagnosis and treatment. Clin Sports Med, 2001. 20(3): p. 565-90.
7. Shimada, K., et al., Reconstruction with an osteochondral autograft for advanced osteochondritis dissecans of the elbow. Clin Orthop Relat Res, 2005(435): p. 140-7.
8. Bauer, M., et al., Osteochondritis dissecans of the elbow. A long-term follow-up study. Clin Orthop Relat Res, 1992(284): p. 156-60.
9. Takahara, M., et al., Nonoperative treatment of osteochondritis dissecans of the humeral capitellum. Am J Sports Med, 1999. 27(6): p. 728-32.
10. Takahara, M., et al., Long term outcome of osteochondritis dissecans of the humeral capitellum. Clin Orthop Relat Res, 1999(363): p. 108-15.
11. Haraldsson, S., On osteochondrosis deformans juvenilis capituli humeri including investigation of intra-osseous vasculature in distal humerus. Acta Ortho Scand, 1959. 38 (suppl): p. 1-232.
12. Pill, S., et al., Role of MRI and clinical criteria in predicting successful nonoperative treatment of osteochondritis dissecans in children. JPO, 2003. 23: p. 102-108.
13. Takahara, M., et al., Classification, treatment, and outcome of osteochondritis dissecans of the humeral capitellum. J Bone Joint Surg Am, 2007. 89(6): p. 1205-14.
14. Takahara, M., et al., Classification, treatment, and outcome os osteochondritis dissecans of the humeral capitellum: Surgical technique. JBJS, 2008. 90(Suppl 2 (Part 1)): p. 47-62.
15. Byrd, J.W.T. and K.S. Jones, Arthroscopic Surgery for Isolated Capitellar Osteochondritis Dissecans in Adolescent Baseball Players: Minimum Three-Year Follow-Up. Am J Sports Med, 2002. 30(4): p. 474-478.
16. Baker, C.L. and A.A. Romeo, Osteochondritis Dissecans of the Capitellum. The American Journal of Sports Medicine: p. -.
17. Jones, K., et al., Arthroscopic management of osteochondritis dissecans of the capitellum: Mid-term results in adolescent athletes. JPO, 2010. 30: p. 8-13.
18. Krijnen, M.R., L. Lim, and W.J. Willems, Arthroscopic treatment of osteochondritis dissecans of the capitellum: Report of 5 female athletes. Arthroscopy, 2003. 19(2): p. 210-4.
19. Mihara, K., et al., Surgical treatment for osteochondritis dissecans of the humeral capitellum. JSES, 2010. 19(1): p. 31-37.
20. Yamamoto, Y., et al., Osteochondral Autograft Transplantation for Osteochondritis Dissecans of the Elbow in Juvenile Baseball Players. The American Journal of Sports Medicine, 2006. 34(5): p. 714-720.

6 Responses to “OCD of the Elbow”

  1. My son was diagnosed last week with severe ocd lesion in his non dominate arm as well as early stage in his right arm. His left arm is stuck at a 90 degree angle because of a broken piece. He will be having open surgery with removal of fragment as well as micro fractures of his left arm. Is there something we are missing. What should we do about the right arm? Looking for care from someone who knows more about it. We will be seeking care at shriners in portland oregon. Is this the best option? Any advice, research etc. would be great. Thanks from a concerned mom.

    • Our son, age 12, had OCD of the right elbow. He had arthroscopic debridement and microfracture on May 1, 2014. He is now eperiencing pain in his left elbow (non-dominant). As of the 6 month check up, his right elbow appears to be doing great. He stayed out of sports for 6 months and just started back to basketball. Before injury, he played baseball, football and basketball. We have an appointment today with his orthopeddic surgeon. I really think there is a genetic component. I worry what the future holds. I hope your sons are doing well and are able to return to activity/sports. Emily or Sonja- how are your sons doing today?

    • Our daughter was just diagnosed with severe OCD in her dominant elbow. She is 12 and a gymnast. Several loose bodies were identified in the MRI. Cannot extend her arm. Been referred to a surgeon who specializes in adolescent sports trauma. We are just trying to learn about the condition and future prognosis

      • Our daughter just had an MRI and is 13 year old gymnast. What have you experienced with this. We meet with doctor and surgeon tomorrow. What type of recovery has she had?

  2. Sonja- our son was diagnosed in 2012 with OCD on his right elbow at 14… had surgery to remove the bone, missed sports for 7 months. He played basketball, baseball, and football at the high school level this past year with zero problems with his right elbow. Unfortunately his left elbow now had OCD. He is 16. We are meeting with the surgeon this week as the bone has broken off. We had no idea his non throwing arm would be at Rick. Hope all is going well with your son. We are hoping this is the last time we have to deal with this.

  3. Great article. 45. 18months ago, while diving in a volleyball tournament for a ball, my elbow experience a direct hit. 8weeks later my elbow, still swollen with fluid, i met with my sports ortho who after xray indicated olecrannon bursitis and the fluid & blood could no longer be removed from elbow. Ace wrap for 1mo, swelling went down. however, since then, elbow has specific spot that I can pinpoint that has never recovered. range of motion at 90% on extension. I have continued to workout, I feel it, i know it is there. Locking, clicking now more frequent. i of course chalk it up to sports athletics, college & pro soccer, baseball, vball, radical snowboarding etc..after reading this well written article and others, i believe it is time to go see the dr. My question is given the length of time since the injury, is it too late to immobilize and treat non-surgically? if immobilize, what position, straight, bent and minimum length of time.



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